References:
1. Gu W, Niu YY, Wang WZ, Liang ZY, Jin XF, Huo H, Wang J.
Analysis of reflux as
the aetiology of laryngeal dysplasia progression through a matched
case-control study. Clin Otolaryngol . 2020. doi:
10.1111/coa.13638.
2. Jiang A, Liang M, Su Z, Chai L, Lei W, Wang Z, Wang A, Wen W, Chen M.
Immunohistochemical
detection of pepsin in laryngeal mucosa for
diagnosing laryngopharyngeal reflux. Laryngoscope . 2011;
121(7):1426-30. doi: 10.1002/lary.21809.
3. Lechien JR, Bobin F, Muls V, Horoi M, Thill MP, Dequanter D, Finck C,
Rodriguez A, Saussez S.
Saliva Pepsin
Concentration of Laryngopharyngeal Reflux Patients Is Influenced
by Meals Consumed Before the Samples. Laryngoscope. 2020. doi:
10.1002/lary.28756.
4. Lechien JR, Bock JM, Carroll TL, Akst LM.
Is empirical treatment
a reasonable strategy for laryngopharyngeal reflux? A contemporary
review. Clin Otolaryngol . 2020; 45(4):450-458. doi:
10.1111/coa.13518.
5. Trivedi S, Rosen CA, Ferris RL.
Current understanding
of the tumor microenvironment of laryngeal dysplasiaand progression to
invasive cancer. Curr Opin Otolaryngol Head Neck Surg . 2016
Apr;24(2):121-7. doi: 10.1097/MOO.0000000000000245.
6. Shellman Z, Aldhahrani A, Verdon B, Mather M, Paleri V, Wilson J,
Pearson J, Ward C, Powell J.
Bile acids: a potential
role in the pathogenesis of pharyngeal malignancy. Clin
Otolaryngol . 2017; 42(5):969-973. doi: 10.1111/coa.12822.