References:
1. Gu W, Niu YY, Wang WZ, Liang ZY, Jin XF, Huo H, Wang J. Analysis of reflux as the aetiology of laryngeal dysplasia progression through a matched case-control study. Clin Otolaryngol . 2020. doi: 10.1111/coa.13638.
2. Jiang A, Liang M, Su Z, Chai L, Lei W, Wang Z, Wang A, Wen W, Chen M. Immunohistochemical detection of pepsin in laryngeal mucosa for diagnosing laryngopharyngeal reflux. Laryngoscope . 2011; 121(7):1426-30. doi: 10.1002/lary.21809.
3. Lechien JR, Bobin F, Muls V, Horoi M, Thill MP, Dequanter D, Finck C, Rodriguez A, Saussez S. Saliva Pepsin Concentration of Laryngopharyngeal Reflux Patients Is Influenced by Meals Consumed Before the Samples. Laryngoscope. 2020. doi: 10.1002/lary.28756.
4. Lechien JR, Bock JM, Carroll TL, Akst LM. Is empirical treatment a reasonable strategy for laryngopharyngeal reflux? A contemporary review. Clin Otolaryngol . 2020; 45(4):450-458. doi: 10.1111/coa.13518.
5. Trivedi S, Rosen CA, Ferris RL. Current understanding of the tumor microenvironment of laryngeal dysplasiaand progression to invasive cancer. Curr Opin Otolaryngol Head Neck Surg . 2016 Apr;24(2):121-7. doi: 10.1097/MOO.0000000000000245.
6. Shellman Z, Aldhahrani A, Verdon B, Mather M, Paleri V, Wilson J, Pearson J, Ward C, Powell J. Bile acids: a potential role in the pathogenesis of pharyngeal malignancy. Clin Otolaryngol . 2017; 42(5):969-973. doi: 10.1111/coa.12822.