Our work was conducted using the model plant Arabidopsis thaliana and published in the journal PLOS Genetics \cite{28937985}. We discovered that the key gene -HsfA2- activates the chaperone synthesis and thus rescues the cell from the toxic effects produced by misfolded protein accumulations. This key gene is also activated when a heat stroke causes problems of protein folding in other cellular compartments. Knowing how plants respond to the challenge of having some of their proteins lose their original structure and function, becoming potentially dangerous, is essential for better crop adaptation to adverse environmental conditions. This challenge is particularly relevant in the current context of climate change.